tag:blogger.com,1999:blog-1568675076199634049Sun, 05 Feb 2012 04:43:01 +0000squamous cell carcinomachronic myeloid leukemia mcqscoronary arteriesrbgamma favre bodiesknudson's hypothesisabnormal hemoglobins mcqsHypersensitivity pneumonitisspheroechinocytepathology mcqsgist prognosisMesotheliomaapoptosisPleural fibrous plaquesenterovirus 70aids related cancersspicules on rbcIg E mcqsBorrelia Burgdoferiaiims pathology past questionsCD4 count decreasesrbc abnormal sizeslibmann sack's endocarditisIg A mcqshemosiderinsilicosisp53chordomaprausnitz kustner reactionthalassemiaaiims november 2008 pathology mcqsgist mcqstypes of bodies in pathologythalassemia rbc changescml mcqshemoglobin kansasPaul Bannel Testtp53 genesereny testMc Conkey mediumburkitt's lymphomaIg M mcqsDimorphic fungiphiladelphia chromosomecowdry type b bodiesProgressive interstitial fibrosisrbc mcqsrespiratory systemAlpha-1-antitrypsin deficiencyAdenocarcinomaprognostic indicators of gastrointestinal stromal tumorsheartrbc pathologykaposi sarcomatwo-hit hypothesisaiims novemeber complete 2008 question paperp27kip1triphasic course of cmlopportunistic infectionsgastrointestinal stromal tumor mcqshemoglobin yakimaskin changes in HIVhair on end appearancemycosis fungoidesimmunology mcqssea blue histiocytesextramedullary hematopoiesisCholerahivtypes of inclusion bodies in pathologynf-2Blastomycosisrbc polymorphismimmunoglobulins mcqscomplement mcqspaget's disease of nippleExtrinsic allergic alveolitisnormal heart external appearancecrenated red cellred blood cell different shapeshairy cell leukemiaburr cellForssman Antigenintracytoplasmic inclusion bodiesPneumococcal PneumoniaAIDSrbc abnormal shapesleft anterior descending coronary arterystomach tumors mcqsmyelofibrosis mcqshemoglobin ecentriacinar emphysemamcqsepicardial surfacetear drop cellcd 117mallory bodypseudo gaucher cellspsammoma bodiesLung abscesshb m iwataBronchiectasistumor suppressor genesaortic rootHistoplasma capsulatumbacillus anthraxhemoglobin kolnwilson's chromosomeIg G mcqsclostridium welchicowdry type a bodiesCytomegalovirusFarmer’s Lungechinocyteantioncogenesirriversible cell injuryakt activationlouis pasteurmarfan's syndromeloa loaIg D mcqsSarcoidosisrb genealizarin redCMVmc callum plaquespathologyrbc disorders smear diagnosisbronchopneumoniakaposi's sarcomaDiffuse alveolar hemorrhageimmunohistochemistry of gistradon exposurewinkler's and ross bodiesStreptococcus pneumoniaeMycobacterium tuberculosisc-kitProzone Phenomenonhemoglobin cpapillary thyroid carcinomaorphan annie nucleusnephritic syndromeemphysemadacrocyteacute myelogenous leukemiaWegener granulomatosishttp://ourpathology.blogspot.com/noreply@blogger.com (doctor)Blogger30125unauthorised copying and publishing of any material from this blog is strictly prohibitedpathology,of,diseases,multiple,choice,questions,human,pathology,mcqsprashanthparigela@gmail.comdoctordoctornopathology,of,diseases,multiple,choice,questions,human,pathology,mcqsPathology Mcqspathology of diseases multiple choice questions human pathology mcqsPathologyMcqshttp://feedburner.google.comtag:blogger.com,1999:blog-1568675076199634049.post-8725585116860028343Thu, 04 Mar 2010 10:56:00 +00002010-03-04T02:59:00.971-08:00*Nephritic syndromes with low complement levels :    (Immune complex glomerulonephritis) - Idiopathic proliferative glomerulonephritis - Crescentric glomerulonephritis - Membranoproliferative glomerulonephritis - Lupus nephritis - Cryoglobulinemia - Bacterial endocarditis - Shunt nephritis - Post infectious glomerulonephritis (post streptococcal) *Nephritic syndromes with Normal complement http://feedproxy.google.com/~r/PathologyMcqs/~3/XVjOrJx7Ezg/29-differential-diagnosis-of-nephritic.htmlprashanthparigela@gmail.com (doctor)1 <p><a href="http://feedads.g.doubleclick.net/~a/eEdafDZbywiXoOrvfkleqoUO1Bo/0/da"><img src="http://feedads.g.doubleclick.net/~a/eEdafDZbywiXoOrvfkleqoUO1Bo/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/eEdafDZbywiXoOrvfkleqoUO1Bo/1/da"><img src="http://feedads.g.doubleclick.net/~a/eEdafDZbywiXoOrvfkleqoUO1Bo/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2010/03/29-differential-diagnosis-of-nephritic.htmltag:blogger.com,1999:blog-1568675076199634049.post-5334600598999795318Sun, 18 Oct 2009 07:29:00 +00002010-02-23T08:32:32.520-08:00types of inclusion bodies in pathologywinkler's and ross bodiescowdry type a bodiescowdry type b bodiestypes of bodies in pathologyintracytoplasmic inclusion bodiesgamma favre bodies <!-- /* Font Definitions */ @font-face {font-family:"Cambria Math"; panose-1:2 4 5 3 5 4 6 3 2 4; mso-font-charset:1; mso-generic-font-family:roman; mso-font-format:other; mso-font-pitch:variable; mso-font-signature:0 0 0 0 0 0;} @font-face {font-family:Calibri; panose-1:2 15 5 2 2 2 4 3 2 4; mso-font-charset:0; http://feedproxy.google.com/~r/PathologyMcqs/~3/qDTnlaymZek/28-types-of-bodies-in-pathology_18.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/7OHfwe-9XG1HZG5eO5d08Cl2lvg/0/da"><img src="http://feedads.g.doubleclick.net/~a/7OHfwe-9XG1HZG5eO5d08Cl2lvg/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/7OHfwe-9XG1HZG5eO5d08Cl2lvg/1/da"><img src="http://feedads.g.doubleclick.net/~a/7OHfwe-9XG1HZG5eO5d08Cl2lvg/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2009/10/28-types-of-bodies-in-pathology_18.htmltag:blogger.com,1999:blog-1568675076199634049.post-4261117356170918912Sat, 17 Oct 2009 12:11:00 +00002009-10-17T05:11:21.426-07:00chronic myeloid leukemia mcqssea blue histiocytespseudo gaucher cellstriphasic course of cmlcml mcqsWhich of the followings are correct about chronic myeloid leukemia? A. It is common in young adults and children. B. It typically takes a biphasic chronic and acute course. C. Pseudo-Gaucher cells are present in the bone marrow. D. Immunohistochemistry for terminal deoxynucleotidyl transferase is a helpful way to confirm the diagnosis. E. Immunohistochemistry for tryptase is a helpful way http://feedproxy.google.com/~r/PathologyMcqs/~3/icvCjiS5Jhg/27-chronic-myeloid-leukemia-cml-mcqs.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/XZ2dr1bTgl_WtOyrw4zTpV9ctcY/0/da"><img src="http://feedads.g.doubleclick.net/~a/XZ2dr1bTgl_WtOyrw4zTpV9ctcY/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/XZ2dr1bTgl_WtOyrw4zTpV9ctcY/1/da"><img src="http://feedads.g.doubleclick.net/~a/XZ2dr1bTgl_WtOyrw4zTpV9ctcY/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2009/10/27-chronic-myeloid-leukemia-cml-mcqs.htmltag:blogger.com,1999:blog-1568675076199634049.post-8788990462259732787Sat, 17 Oct 2009 12:05:00 +00002009-10-17T12:03:56.718-07:00immunohistochemistry of gistgastrointestinal stromal tumor mcqsc-kitcd 117prognostic indicators of gastrointestinal stromal tumorsgist mcqsstomach tumors mcqsgist prognosis1.Which of the following(s) is/are true about gastrointestinal stromal tumor (GIST): 1. Most commonly seen in small intestine. 2. The most common histopathologic picture is that of spindle cells and epithelioid cells arranged in storiform pattern. 3. Common in young patients under 30 years of age. 4. Positive for CD117 (c-kit) and CD34. A. 1, 2, and 3 are correct. B. 1 and 3 are correcthttp://feedproxy.google.com/~r/PathologyMcqs/~3/LTZekx4rPlI/26-gastrointestinal-stromal-tumor-gist.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/tyL-2QxfvgM-FZGpYzoFFpUFwCE/0/da"><img src="http://feedads.g.doubleclick.net/~a/tyL-2QxfvgM-FZGpYzoFFpUFwCE/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/tyL-2QxfvgM-FZGpYzoFFpUFwCE/1/da"><img src="http://feedads.g.doubleclick.net/~a/tyL-2QxfvgM-FZGpYzoFFpUFwCE/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2009/10/26-gastrointestinal-stromal-tumor-gist.htmltag:blogger.com,1999:blog-1568675076199634049.post-280253445151146298Fri, 29 May 2009 16:23:00 +00002009-05-29T09:23:39.126-07:00akt activationtp53 genep27kip1two-hit hypothesistumor suppressor genesknudson's hypothesisantioncogenesrb geneA tumor suppressor gene, or antioncogene is a gene that protects a cell from one step on the path to cancer. When this gene is mutated to cause a loss or reduction in its function, the cell can progress to cancer, usually in combination with other genetic changes. TWO-HIT HYPOTHESIS : Unlike oncogenes, tumor suppressor genes generally follow the 'two-hit hypothesis', which implies that both http://feedproxy.google.com/~r/PathologyMcqs/~3/r4zEs9h6yBY/25-antioncogenes-tumor-suppressor-genes.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/xy--dkZORbdcsKuof6i4Bl5K4TI/0/da"><img src="http://feedads.g.doubleclick.net/~a/xy--dkZORbdcsKuof6i4Bl5K4TI/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/xy--dkZORbdcsKuof6i4Bl5K4TI/1/da"><img src="http://feedads.g.doubleclick.net/~a/xy--dkZORbdcsKuof6i4Bl5K4TI/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2009/05/25-antioncogenes-tumor-suppressor-genes.htmltag:blogger.com,1999:blog-1568675076199634049.post-1921179106264898556Thu, 12 Mar 2009 07:43:00 +00002009-03-12T01:14:28.640-07:00hemoglobin ehemoglobin yakimahb m iwatahemoglobin kolnhemoglobin chemoglobin kansasabnormal hemoglobins mcqs1q: what is the hemoglobin that results when glutamate in the 6th position of beta chain is replaced by valine ?a. hemoglobin S or sickle cell hemoglobinb. hemoglobin Cc. hemoglobin Ed. hemoblogin Yakima answer a . sickle cell hemoglobin also called as hemoglobin S . this is mostly seen in African population and is characterized by anemia and ischemic infarcts . 2q: what is the hemoglobin http://feedproxy.google.com/~r/PathologyMcqs/~3/x1GtUHLi4h0/25-abnormal-hemoglobins-with-altered.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/cbd8HZXPI_YoVDlEAsc2fCl4wzY/0/da"><img src="http://feedads.g.doubleclick.net/~a/cbd8HZXPI_YoVDlEAsc2fCl4wzY/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/cbd8HZXPI_YoVDlEAsc2fCl4wzY/1/da"><img src="http://feedads.g.doubleclick.net/~a/cbd8HZXPI_YoVDlEAsc2fCl4wzY/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2009/03/25-abnormal-hemoglobins-with-altered.htmltag:blogger.com,1999:blog-1568675076199634049.post-7915871941560527431Sat, 15 Nov 2008 09:02:00 +00002008-11-15T01:07:15.272-08:00aiims november 2008 pathology mcqspathology mcqsaiims pathology past questionsaiims novemeber complete 2008 question paper<!--[if gte mso 9]> Normal 0 MicrosoftInternetExplorer4 <![endif]--><!--[if !mso]> st1\:*{behavior:url(#ieooui) } <![endif]--> <!-- /* Style Definitions */ p.MsoNormal, li.MsoNormal, div.MsoNormal {mso-style-parent:""; margin:0in; margin-bottom:.0001pt; mso-pagination:widow-orphan; font-size:12.0pt; font-family:"Times New Roman"; mso-fareast-font-family:http://feedproxy.google.com/~r/PathologyMcqs/~3/xiI8R9J3A4k/24-aiims-november-2008-pathology-mcqs.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/TBRIeCD7bpTjkiBCyMeoFN46WNg/0/da"><img src="http://feedads.g.doubleclick.net/~a/TBRIeCD7bpTjkiBCyMeoFN46WNg/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/TBRIeCD7bpTjkiBCyMeoFN46WNg/1/da"><img src="http://feedads.g.doubleclick.net/~a/TBRIeCD7bpTjkiBCyMeoFN46WNg/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/11/24-aiims-november-2008-pathology-mcqs.htmltag:blogger.com,1999:blog-1568675076199634049.post-1437596858390668949Mon, 22 Sep 2008 18:10:00 +00002008-09-22T11:15:33.535-07:00Ig D mcqsIg A mcqsIg M mcqscomplement mcqsIg E mcqsIg G mcqsimmunoglobulins mcqsimmunology mcqsprausnitz kustner reaction1. Ig that crosses the placenta ?<!--[endif]--> Answer: Ig G . <!--[if !supportLists]-->2. Ig present in milk ?<!--[endif]--> Answer: Ig G and Ig A <!--[if !supportLists]-->3. Ig in seromucinous glands ?<!--[endif]--> Answer: Ig G and Ig A <!--[if !supportLists]-->4. Ig in primary immune response ?<!--[endif]--> Answer: Ig M <!--[if !http://feedproxy.google.com/~r/PathologyMcqs/~3/RdYiLaCe-CY/23-immunoglobulins-mcqs-and-faqs.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/K25zhgnKF4EVHnK3OiyOsIVCce0/0/da"><img src="http://feedads.g.doubleclick.net/~a/K25zhgnKF4EVHnK3OiyOsIVCce0/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/K25zhgnKF4EVHnK3OiyOsIVCce0/1/da"><img src="http://feedads.g.doubleclick.net/~a/K25zhgnKF4EVHnK3OiyOsIVCce0/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/09/23-immunoglobulins-mcqs-and-faqs.htmltag:blogger.com,1999:blog-1568675076199634049.post-3932505413649488878Mon, 08 Sep 2008 02:48:00 +00002008-09-07T19:53:23.393-07:00 Evolution of Morphologic Changes in Myocardial Infarction Time Gross Features Light Microscopic Findings 0-½ hr None None ½-4hr None Usually http://feedproxy.google.com/~r/PathologyMcqs/~3/7T095xsXhyk/22-morphological-changes-in-myocardial.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/bq5TiY4Ml-bT9ywvVZvcTo-lE80/0/da"><img src="http://feedads.g.doubleclick.net/~a/bq5TiY4Ml-bT9ywvVZvcTo-lE80/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/bq5TiY4Ml-bT9ywvVZvcTo-lE80/1/da"><img src="http://feedads.g.doubleclick.net/~a/bq5TiY4Ml-bT9ywvVZvcTo-lE80/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/09/22-morphological-changes-in-myocardial.htmltag:blogger.com,1999:blog-1568675076199634049.post-6360153108383822928Tue, 08 Jul 2008 19:53:00 +00002008-07-08T12:57:25.590-07:00rbc abnormal shapesspicules on rbccrenated red cellrbc abnormal sizesrbc polymorphismechinocyterbc disorders smear diagnosisburr cellspheroechinocyterbc mcqs Definition: Red cell membrane abnormality characterized by short, blunt projections (spicules) of relatively uniform length and evenly distributed over the red cell surface. In early stage of development the red cell maintains a discoid shape (echinocyte I), but may progress to a spherical shape (echinocyte IV, spheroechinocyte). Pathobiology: Due to an expansion of the outer leaflet of http://feedproxy.google.com/~r/PathologyMcqs/~3/kyJMQbt7za0/21-echinocyte-burr-cell.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/I40ZsODy5-7xA85Qv8-WejDb51M/0/da"><img src="http://feedads.g.doubleclick.net/~a/I40ZsODy5-7xA85Qv8-WejDb51M/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/I40ZsODy5-7xA85Qv8-WejDb51M/1/da"><img src="http://feedads.g.doubleclick.net/~a/I40ZsODy5-7xA85Qv8-WejDb51M/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/07/21-echinocyte-burr-cell.htmltag:blogger.com,1999:blog-1568675076199634049.post-3431339763667766496Tue, 08 Jul 2008 19:24:00 +00002008-07-08T12:29:14.412-07:00rbc abnormal shapestear drop cellred blood cell different shapesmyelofibrosis mcqsthalassemia rbc changesrbc pathologyrbc polymorphismextramedullary hematopoiesisdacrocyterbc mcqsDacryocyte is a pear-shaped or tear-shaped red blood cell that is usually seen in a broad spectrum of infiltrative disorders that disrupt the bone marrow architecture and also in extramedullary hematopoiesis.hence it is called TEAR DROP CELL .The abnormality is thought to be due to membrane damage to the red cell during maturation in a crowded bone marrow environment, damage at the time of exit http://feedproxy.google.com/~r/PathologyMcqs/~3/j9qk_kwOBOg/20-dacrocyte.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/the5mwfKpKL2-4Fgdoiu9JJnd5k/0/da"><img src="http://feedads.g.doubleclick.net/~a/the5mwfKpKL2-4Fgdoiu9JJnd5k/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/the5mwfKpKL2-4Fgdoiu9JJnd5k/1/da"><img src="http://feedads.g.doubleclick.net/~a/the5mwfKpKL2-4Fgdoiu9JJnd5k/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/07/20-dacrocyte.htmltag:blogger.com,1999:blog-1568675076199634049.post-3063037005265244171Thu, 20 Mar 2008 11:25:00 +00002008-03-20T04:28:00.261-07:00hivaids related cancersCMVopportunistic infectionsskin changes in HIVCD4 count decreaseskaposi's sarcomaAIDSkaposi sarcoma Kaposi's sarcoma typically produces one or more reddish purple nodules on the skin, as seen here grossly.http://feedproxy.google.com/~r/PathologyMcqs/~3/_6BjnT9CQm8/19-kaposi-sarcoma.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/hgjsVDxeGM8HReWhokcy4CKrBpY/0/da"><img src="http://feedads.g.doubleclick.net/~a/hgjsVDxeGM8HReWhokcy4CKrBpY/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/hgjsVDxeGM8HReWhokcy4CKrBpY/1/da"><img src="http://feedads.g.doubleclick.net/~a/hgjsVDxeGM8HReWhokcy4CKrBpY/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/03/19-kaposi-sarcoma.htmltag:blogger.com,1999:blog-1568675076199634049.post-5506149738654641311Thu, 20 Mar 2008 06:23:00 +00002008-03-19T23:25:49.219-07:00heartcoronary arteriesaortic rootleft anterior descending coronary arterynormal heart external appearanceepicardial surface This is the external appearance of a normal heart.The epicardial surface is smooth and glistening.The amount of epicardial fat is usual.The left anterior descending coronary artery extends down from the aortic root to the apex.http://feedproxy.google.com/~r/PathologyMcqs/~3/zJhvp-0Fdi4/18-normal-heart.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/B5z97qCuJmSjwaE5OMDLazJQD4c/0/da"><img src="http://feedads.g.doubleclick.net/~a/B5z97qCuJmSjwaE5OMDLazJQD4c/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/B5z97qCuJmSjwaE5OMDLazJQD4c/1/da"><img src="http://feedads.g.doubleclick.net/~a/B5z97qCuJmSjwaE5OMDLazJQD4c/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/03/18-normal-heart.htmltag:blogger.com,1999:blog-1568675076199634049.post-991570791497820278Mon, 14 Jan 2008 14:04:00 +00002008-01-23T07:40:47.028-08:00Borrelia BurgdoferiMc Conkey mediumFarmer’s LungpathologymcqsCholera71) Urease producing all except a. H. pylori b. Salmonella c. Klebsilla d. proteus<?xml:namespace prefix = o />Answer : b) SalmonellaReference: Microbiology 7th Edition Pages 290 72) Vibrio Cholera usually is transported using a. VR Medium b. BSA Medium c. GTTA Medium d. None of the aboveAnswer : a) VR mediumReference: Microbiology 7th Edition Page 306 73) An example of Double http://feedproxy.google.com/~r/PathologyMcqs/~3/ShbF1Rj59nc/17-pathology-mcqs-71-to-80.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/YOWF7QxYeiKxADEzJcmDJzeOAvg/0/da"><img src="http://feedads.g.doubleclick.net/~a/YOWF7QxYeiKxADEzJcmDJzeOAvg/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/YOWF7QxYeiKxADEzJcmDJzeOAvg/1/da"><img src="http://feedads.g.doubleclick.net/~a/YOWF7QxYeiKxADEzJcmDJzeOAvg/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/17-pathology-mcqs-71-to-80.htmltag:blogger.com,1999:blog-1568675076199634049.post-2775997377877751532Mon, 14 Jan 2008 14:00:00 +00002008-01-23T07:46:01.046-08:00sereny testloa loabacillus anthraxenterovirus 70pathologylouis pasteurmcqs61) Reactivity of false positive VDRL in Acute Malaria is a. 10 to 20 % b. 10 to 30 % c. 50 % d. 100 %<?xml:namespace prefix = o />Answer : ?? 62) Calabar Swelling is seen in a. loa loa b. Manso c. Wucherchia bancrofti d. Brugui malayiAnswer : a) Loa loaReference: <?xml:namespace prefix = st1 />Harrison 15th Edition Chapter 221 63) Bacteria in air first demonstrated by a. Louis http://feedproxy.google.com/~r/PathologyMcqs/~3/tUmaScblpmI/16-pathology-mcqs-61-to-70.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/hOGMRicna6E0Y5T6oOkAXlNlxas/0/da"><img src="http://feedads.g.doubleclick.net/~a/hOGMRicna6E0Y5T6oOkAXlNlxas/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/hOGMRicna6E0Y5T6oOkAXlNlxas/1/da"><img src="http://feedads.g.doubleclick.net/~a/hOGMRicna6E0Y5T6oOkAXlNlxas/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/16-pathology-mcqs-61-to-70.htmltag:blogger.com,1999:blog-1568675076199634049.post-4042346042742112348Mon, 14 Jan 2008 13:54:00 +00002008-01-23T07:46:31.829-08:00Pneumococcal Pneumoniaclostridium welchiProzone PhenomenonpathologyPaul Bannel Test51) Hepatitis B is aa. Single Stranded DNA Virusb. Double Stranded DNA Virusc. Single Stranded RNA Virusd. Double Stranded RNA Virus <?xml:namespace prefix = o />Answer : (B) Double Stranded DNA VirusRefernce: Ananthanarayanan 7th Edition Page 550Ä All DNA Viruses are Double Stranded except Parvovirus which is a Single stranded DNA VirusÄ All RNA Viruses are Single Stranded except Reovirus which http://feedproxy.google.com/~r/PathologyMcqs/~3/eVXVC7bWjMc/15-pathology-mcqs-51-to-60.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/frnYZl1aU5qzjQqXnpf9A0DE-Us/0/da"><img src="http://feedads.g.doubleclick.net/~a/frnYZl1aU5qzjQqXnpf9A0DE-Us/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/frnYZl1aU5qzjQqXnpf9A0DE-Us/1/da"><img src="http://feedads.g.doubleclick.net/~a/frnYZl1aU5qzjQqXnpf9A0DE-Us/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/15-pathology-mcqs-51-to-60.htmltag:blogger.com,1999:blog-1568675076199634049.post-2088840888773915288Mon, 14 Jan 2008 13:41:00 +00002008-01-22T23:20:28.904-08:00Forssman AntigenHistoplasma capsulatumpathologyDimorphic fungi41 - Allograft is a graft obtained froma. Selfb. Identical Twinc. A member of same species d. A member of other species<?xml:namespace prefix = o />Answer C) A member of same species Reference : Ananthanarayanan 7th Edition Page 177 and Bailey and Love 24th Edition Page <?xml:namespace prefix = st1 />184 Box 14.142 - Acute Epiglottitis is caused bya. H. Influenzaeb. Pneumoccocusc. Beta hemolytic http://feedproxy.google.com/~r/PathologyMcqs/~3/V3wZek2qH_Y/14-pathology-mcqs-41-to-50.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/0I99wZuuw38OZU7B0mD_x0TSwA4/0/da"><img src="http://feedads.g.doubleclick.net/~a/0I99wZuuw38OZU7B0mD_x0TSwA4/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/0I99wZuuw38OZU7B0mD_x0TSwA4/1/da"><img src="http://feedads.g.doubleclick.net/~a/0I99wZuuw38OZU7B0mD_x0TSwA4/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/14-pathology-mcqs-41-to-50.htmltag:blogger.com,1999:blog-1568675076199634049.post-4824395525741983719Mon, 14 Jan 2008 13:18:00 +00002008-01-22T22:48:38.383-08:00wilson's chromosomep53thalassemianf-2rbhair on end appearancenephritic syndrome31) Primary amyloidosis is least common in a. GIT b. Heart c. Liver d. Peripheral nerve<?xml:namespace prefix = o />Answer : c) LiverReference: <?xml:namespace prefix = st1 />Harrison 15th Edition Page 319 32) Heart failure cells are seen in a. Chronic Venous congestion of Lung b. Chronic Venous congestion of Liver c. After infarction in Myocardium d. AortaAnswer : a) Chronic Venous http://feedproxy.google.com/~r/PathologyMcqs/~3/3qr8eAZIlAo/13-pathology-mcqs-31-to-40.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/hWlCItjjKbP0w9hi4L7FQl1KwYg/0/da"><img src="http://feedads.g.doubleclick.net/~a/hWlCItjjKbP0w9hi4L7FQl1KwYg/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/hWlCItjjKbP0w9hi4L7FQl1KwYg/1/da"><img src="http://feedads.g.doubleclick.net/~a/hWlCItjjKbP0w9hi4L7FQl1KwYg/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/13-pathology-mcqs-31-to-40.htmltag:blogger.com,1999:blog-1568675076199634049.post-6054365732597870571Mon, 14 Jan 2008 13:11:00 +00002008-01-22T22:43:26.701-08:00irriversible cell injuryburkitt's lymphomalibmann sack's endocarditisphiladelphia chromosomemc callum plaquesapoptosispathologymcqs21) Howell Jolly bodies are commonly seen in a. Megaloblastic Anemia b. Sickle cell anemia c. Iron deficiency anemia d. After splenectomy<?xml:namespace prefix = o />Answer : d) After Splenectomy Reference: <?xml:namespace prefix = st1 />Harrison 15th Edition Chapter 116 22) Most specifice antibody for SLE a. Anti dsDNA b. SS-A (Ro) c. SS-B (La) d. AnticentromereAnswer : a) Anti ds http://feedproxy.google.com/~r/PathologyMcqs/~3/Vd7JDW9FFII/12-pathology-mcqs-21-to-30.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/kJ-u-dqMqs0uhS5w9idfPlfmJCA/0/da"><img src="http://feedads.g.doubleclick.net/~a/kJ-u-dqMqs0uhS5w9idfPlfmJCA/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/kJ-u-dqMqs0uhS5w9idfPlfmJCA/1/da"><img src="http://feedads.g.doubleclick.net/~a/kJ-u-dqMqs0uhS5w9idfPlfmJCA/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/12-pathology-mcqs-21-to-30.htmltag:blogger.com,1999:blog-1568675076199634049.post-2108404463410700293Mon, 14 Jan 2008 13:08:00 +00002008-01-22T21:24:49.725-08:00psammoma bodiesmallory bodycentriacinar emphysemaorphan annie nucleuspapillary thyroid carcinoma11 - Orphan annie nucleus is seen in a. Papillary Thyroid Carcinomab. Chronic Myeloid Leukemiac. Malignant melanomad. LipomaAnswer (a) Papillary Thyroid CarcinomaReference: Robbins 7th Edition Page 11712 - Mallory body is aa. Keratin<?xml:namespace prefix = o />b. Neurofilamentc. Lipidd. None of the aboveAnswer : (a) keratinReference: Robbins 7th edition Page 3413 - Psammoma bodies are a. http://feedproxy.google.com/~r/PathologyMcqs/~3/RipjoYRvBBI/11-pathology-mcqs-11-to-20.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/_8JSpHZGmWIvc7nZWKfWHOzDx6A/0/da"><img src="http://feedads.g.doubleclick.net/~a/_8JSpHZGmWIvc7nZWKfWHOzDx6A/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/_8JSpHZGmWIvc7nZWKfWHOzDx6A/1/da"><img src="http://feedads.g.doubleclick.net/~a/_8JSpHZGmWIvc7nZWKfWHOzDx6A/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/11-pathology-mcqs-11-to-20.htmltag:blogger.com,1999:blog-1568675076199634049.post-7378909999792104035Mon, 14 Jan 2008 13:00:00 +00002008-01-22T21:12:53.440-08:00chordomahairy cell leukemiamarfan's syndromealizarin redmycosis fungoideshemosiderinpaget's disease of nipple1 - Amyloid is stained bya. Congo redb. Eosinc. Hematozylind. ALIZARIN RED<?xml:namespace prefix = o />Answer (a) Congo RedReference: Robbins 7th Edition Page 2632 - Prussian blue is used for staininga. Haemosiderinb. Bonec. Muscled. FatAnswer : (a) HemosiderinReference: Robbins 7th Edition Page 9093 - Physaliferous Cells are Seen Ina. Chordomab. Lipomac. Neuromad. MeningiomaAnswer : (A) http://feedproxy.google.com/~r/PathologyMcqs/~3/IMvDuUpOqv0/10-pathology-mcqs-1-to-10.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/0oDWR9lmRCFMANesT9wQkB1WPlE/0/da"><img src="http://feedads.g.doubleclick.net/~a/0oDWR9lmRCFMANesT9wQkB1WPlE/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/0oDWR9lmRCFMANesT9wQkB1WPlE/1/da"><img src="http://feedads.g.doubleclick.net/~a/0oDWR9lmRCFMANesT9wQkB1WPlE/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/10-pathology-mcqs-1-to-10.htmltag:blogger.com,1999:blog-1568675076199634049.post-3931102069357646246Sun, 13 Jan 2008 10:22:00 +00002008-01-13T02:25:37.312-08:00Diffuse alveolar hemorrhageWegener granulomatosisrespiratory systempathologymcqsBlastomycosisQuestion 41A 30-year-old woman is in the 28th week of an uncomplicated pregnancy when she experiences the sudden onset of severe abdominal pain, followed by vaginal bleeding, then the onset of labor. A girl infant is delivered on the way to the hospital. On arrival within an hour, the baby is in respiratory distress and requires intubation and mechanical ventilation. A day later, a chest http://feedproxy.google.com/~r/PathologyMcqs/~3/Zchl8Q0WPC4/9-respiratory-system-mcqs-41-to-45.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/vJ6tGILVuTAtyxDIXAnqQKYT6Gg/0/da"><img src="http://feedads.g.doubleclick.net/~a/vJ6tGILVuTAtyxDIXAnqQKYT6Gg/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/vJ6tGILVuTAtyxDIXAnqQKYT6Gg/1/da"><img src="http://feedads.g.doubleclick.net/~a/vJ6tGILVuTAtyxDIXAnqQKYT6Gg/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/9-respiratory-system-mcqs-41-to-45.htmltag:blogger.com,1999:blog-1568675076199634049.post-2860793000438327665Sun, 13 Jan 2008 10:18:00 +00002008-01-13T02:22:05.670-08:00emphysemarespiratory systempathologymcqsAlpha-1-antitrypsin deficiencyQuestion 36A 44-year-old previously healthy man has the sudden onset of severe dyspnea. On physical examination he is afebrile. There are absent breath sounds over the right lung fields. A chest x-ray shows pulmonary atelectasis involving all of the right lung. Which of the following conditions is most likely to produce these findings?A Aspiration of a foreign bodyB Pulmonary embolismC Squamous http://feedproxy.google.com/~r/PathologyMcqs/~3/pLBWyiOVIMA/8-respiratory-system-mcqs-36-to-40.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/Q6PqMehz1tQaBdtpLQY2mU5ZeJw/0/da"><img src="http://feedads.g.doubleclick.net/~a/Q6PqMehz1tQaBdtpLQY2mU5ZeJw/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/Q6PqMehz1tQaBdtpLQY2mU5ZeJw/1/da"><img src="http://feedads.g.doubleclick.net/~a/Q6PqMehz1tQaBdtpLQY2mU5ZeJw/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/8-respiratory-system-mcqs-36-to-40.htmltag:blogger.com,1999:blog-1568675076199634049.post-4288327333788659458Sun, 13 Jan 2008 10:14:00 +00002008-01-13T02:17:14.011-08:00Streptococcus pneumoniaerespiratory systemLung abscesspathologymcqsbronchopneumoniaQuestion 31A 38-year-old previously healthy woman has had a worsening non-productive cough for the past 4 days. On physical examination her temperature is 38.3 C. A chest radiograph shows patchy infiltrates and diffuse interstitial markings. Laboratory studies show a sputum gram stain with mixed flora. Her Hgb is 12.9 g/dL, platelet count 229,450/microliter, and WBC count 5815/microliter. Her http://feedproxy.google.com/~r/PathologyMcqs/~3/OR6uhpN4NaU/7-respiratory-system-mcqs-31-to-35.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/Wm_amNWD3nr2nWEhmpnP54f_4rU/0/da"><img src="http://feedads.g.doubleclick.net/~a/Wm_amNWD3nr2nWEhmpnP54f_4rU/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/Wm_amNWD3nr2nWEhmpnP54f_4rU/1/da"><img src="http://feedads.g.doubleclick.net/~a/Wm_amNWD3nr2nWEhmpnP54f_4rU/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/7-respiratory-system-mcqs-31-to-35.htmltag:blogger.com,1999:blog-1568675076199634049.post-286160351281398375Sun, 13 Jan 2008 10:11:00 +00002008-01-13T02:14:13.977-08:00Mesotheliomarespiratory systemProgressive interstitial fibrosispathologymcqsQuestion 26A 40-year-old previously healthy woman, a non-smoker, has had episodes of fever, non-productive cough, and dyspnea over the past 3 months. Her symptoms disappeared after a month's vacation, but reappeared when she returned home to take care of her canaries. On physical examination there are no abnormal findings. A chest radiograph shows fine diffuse and nodular infiltrates in all lung http://feedproxy.google.com/~r/PathologyMcqs/~3/aQyiIdsqBlU/6-respiratory-system-mcqs-26-to-30.htmlprashanthparigela@gmail.com (doctor)0 <p><a href="http://feedads.g.doubleclick.net/~a/ILqLRTGTpBGwuDZGn-IysC4FFWY/0/da"><img src="http://feedads.g.doubleclick.net/~a/ILqLRTGTpBGwuDZGn-IysC4FFWY/0/di" border="0" ismap="true"></img></a><br/> <a href="http://feedads.g.doubleclick.net/~a/ILqLRTGTpBGwuDZGn-IysC4FFWY/1/da"><img src="http://feedads.g.doubleclick.net/~a/ILqLRTGTpBGwuDZGn-IysC4FFWY/1/di" border="0" ismap="true"></img></a></p>http://ourpathology.blogspot.com/2008/01/6-respiratory-system-mcqs-26-to-30.htmlen-usunauthorised copying and publishing of any material from this blog is strictly prohibiteddoctornonadultPathology Mcqs