<?xml version="1.0" encoding="UTF-8" standalone="no"?><rss xmlns:atom="http://www.w3.org/2005/Atom" xmlns:blogger="http://schemas.google.com/blogger/2008" xmlns:gd="http://schemas.google.com/g/2005" xmlns:georss="http://www.georss.org/georss" xmlns:itunes="http://www.itunes.com/dtds/podcast-1.0.dtd" xmlns:openSearch="http://a9.com/-/spec/opensearchrss/1.0/" xmlns:thr="http://purl.org/syndication/thread/1.0" version="2.0"><channel><atom:id>tag:blogger.com,1999:blog-2649822511498143527</atom:id><lastBuildDate>Sat, 07 Sep 2024 01:09:57 +0000</lastBuildDate><category>osmolality article</category><category>central nervous system physiology mcqs</category><category>osmolality mcqs</category><category>aiims may 2005 physiology mcqs with answers</category><category>mcqs</category><category>physiology</category><category>aiims physiology mcqs</category><category>faecal osmolality</category><category>ganong</category><category>hyponatremia investigation</category><category>osmolality and kidney 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factor</category><category>hematology mcqs</category><category>hormonal control of gastrointestinal motility</category><category>hormones which act through the phopholipase c system</category><category>insulin physiology mcqs</category><category>kinocilium</category><category>law of projection</category><category>lung physiology mcqs</category><category>measurement of body fluid volumes</category><category>mechanism of action of hormones</category><category>myosin</category><category>neocerebellum</category><category>nocturnal penile erection</category><category>nocturnal penile tumescence</category><category>normal pressures in the heart and great vessels</category><category>normal pressures of atria and ventricles</category><category>normal pressures of pulmonary artery</category><category>normal pressures of the cardiovascular system</category><category>npt</category><category>osmolality and ADH</category><category>osmolality and ADH physiology</category><category>osmolality and ADH relation</category><category>osmolality calculating formula</category><category>osmolality clinical uses</category><category>osmolality measurement</category><category>osmolality uses</category><category>osmolar gap identification</category><category>osmometer</category><category>osmotic diarrhea</category><category>oxygen hemoglobin dissociation curve mcqs</category><category>parts of cerebellum</category><category>pgi chandigarh 2001 physiology mcqs</category><category>pgimer chandigarh 2001 december paper mcqs with answers</category><category>phantom limb</category><category>physiology aiims past papers</category><category>physiology mcqs</category><category>physiology mcqs from past aiims papers</category><category>physiology mcqs from past medical pg entrance papers</category><category>physiology mock test 1</category><category>prostate mcqs</category><category>prothrombin time</category><category>regulation of insulin secretion</category><category>rem sleep and wakefulness differences</category><category>renal clearance</category><category>renal failure mcqs</category><category>respiratory system physiology mcqs</category><category>right parietal lobe</category><category>right ventricular pressure</category><category>second messengers mcqs</category><category>secondary messenger systems</category><category>secretin actions</category><category>sensory dermatomes</category><category>serum osmolality</category><category>serum osmolality calculation formula</category><category>serum osmolar gap</category><category>siadh identification</category><category>simethicone</category><category>spinocerebellum</category><category>stereocilia</category><category>substantia gelatinosa rolando</category><category>sympathetic nervous system</category><category>transportor binding proteins</category><category>tropomyosin</category><category>troponin</category><category>true hyponatremia</category><category>units of osmolality</category><category>v1a receptor</category><category>v1b receptor</category><category>v2 receptor</category><category>vanilloid receptors</category><category>vasopressin action on various organs</category><category>vasopressin receptors</category><category>veins mcqs</category><category>vestibulo-cerebellum</category><category>wernicke's aphasia</category><title>Human Physiology Mcqs Postgraduation Entrance preparation</title><description></description><link>http://ourphysiology.blogspot.com/</link><managingEditor>noreply@blogger.com (Unknown)</managingEditor><generator>Blogger</generator><openSearch:totalResults>36</openSearch:totalResults><openSearch:startIndex>1</openSearch:startIndex><openSearch:itemsPerPage>25</openSearch:itemsPerPage><language>en-us</language><itunes:explicit>no</itunes:explicit><copyright>unauthorised copying and publishing of any material from this blog is strictly prohibited</copyright><itunes:keywords>human,physiology,multiple,choice,questions,mcqs,on,human,organ,systems,functioning,physiology</itunes:keywords><itunes:summary>human physiology multiple choice questions mcqs on human organ systems functioning physiology </itunes:summary><itunes:subtitle>human physiology mcqs</itunes:subtitle><itunes:category text="Education"/><itunes:author>doctor</itunes:author><itunes:owner><itunes:email>prashanthparigela@gmail.com</itunes:email><itunes:name>doctor</itunes:name></itunes:owner><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6324694409904165307</guid><pubDate>Fri, 21 May 2010 09:46:00 +0000</pubDate><atom:updated>2010-05-21T02:46:50.440-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">aiims may 2010 physiology mcqs with answers</category><category domain="http://www.blogger.com/atom/ns#">aiims past papers physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">physiology mcqs from past aiims papers</category><title>36 - AIIMS May 2010 Physiology Mcqs</title><atom:summary type="text">16. Appetite is stimulated by A/E
a) Agouti related peptide
b) Melanocyte concentrating hormone
c) Melanocyte stimulating hormone
d) Neuropeptide Y

answer c. Melanocyte stimulating hormone. 
17. Capacitation occurs in
a) Uterus
b) Seminal vesicle
c) Epididymis
d) Vas deferens

answer a. Uterus. 
18. The main cause of increased blood flow to exercising muscles is
a) Raised blood pressure
b) </atom:summary><link>http://ourphysiology.blogspot.com/2010/05/36-aiims-may-2010-physiology-mcqs.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-1282743123614907131</guid><pubDate>Thu, 04 Mar 2010 06:39:00 +0000</pubDate><atom:updated>2010-03-03T22:42:53.996-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">dap/ip3 second messenger system hormones</category><category domain="http://www.blogger.com/atom/ns#">hormones which act through the phopholipase c system</category><category domain="http://www.blogger.com/atom/ns#">mechanism of action of hormones</category><category domain="http://www.blogger.com/atom/ns#">peptide hormones mcqs</category><category domain="http://www.blogger.com/atom/ns#">secondary messenger systems</category><title>35 - Second messengers</title><atom:summary type="text">


</atom:summary><link>http://ourphysiology.blogspot.com/2010/03/35-second-messengers.html</link><media:thumbnail xmlns:media="http://search.yahoo.com/mrss/" height="72" url="https://blogger.googleusercontent.com/img/b/R29vZ2xl/AVvXsEi-KAIc0Je92hyphenhyphenOBHwZOWS_KpeWdE83A4OxkuGOF3WBS4I7dWvWTkkPswxLBW_SPD3QCTH1f0vEAUn1pCp0nNeUIN_OQWKn4HLbT2yGmPkc5B-QPD1TGvmbtkKdDvldDdGPjr_u9UIKVUY/s72-c/Second_messenger_cyclic_amp_adenylyl_cyclase1.jpg" width="72"/><thr:total>2</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-8893391493688643051</guid><pubDate>Mon, 22 Feb 2010 11:37:00 +0000</pubDate><atom:updated>2010-02-23T08:16:51.700-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">factors decreasing insulin secretion</category><category domain="http://www.blogger.com/atom/ns#">factors increasing insulin secretion</category><category domain="http://www.blogger.com/atom/ns#">factors regulating insulin secretion</category><category domain="http://www.blogger.com/atom/ns#">regulation of insulin secretion</category><title>34 - Factors controlling Insulin secretion</title><atom:summary type="text">*FACTORS THAT INCREASE INSULIN SECRETION:
- Increase in blood glucose
- Increase in blood free fatty acids
- Increase in blood amino acids
- Gastrointestinal hormones (Gastrin, CCK, Secretin and GIP)
- Glucagon, GH, Cortisol
- Parasympathetic stimulation; Acetyl choline
- Beta-adrenergic stimulation
- Insulin resistance; Obesity
- Sulfonyl urea drugs (Glyburide, Tolbutamide)

*FACTORS THAT </atom:summary><link>http://ourphysiology.blogspot.com/2010/02/34-factors-controlling-insulin.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-2608219251722156141</guid><pubDate>Mon, 22 Feb 2010 10:10:00 +0000</pubDate><atom:updated>2010-02-22T02:11:16.718-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">cholecystokinin</category><category domain="http://www.blogger.com/atom/ns#">cholegogues</category><category domain="http://www.blogger.com/atom/ns#">cholerectics</category><category domain="http://www.blogger.com/atom/ns#">factors influencing gastric emptying</category><category domain="http://www.blogger.com/atom/ns#">gastrin</category><category domain="http://www.blogger.com/atom/ns#">hormonal control of gastrointestinal motility</category><category domain="http://www.blogger.com/atom/ns#">secretin actions</category><title>33 - Hormonal control of Gastrointestinal motility</title><atom:summary type="text">*Gastrin is secreted by the “G” cells of the antrum of&amp;nbsp;the stomach in response to stimuli associated with
ingestion of a meal, such as distention of the stomach,&amp;nbsp;the products of proteins, and gastrin releasing peptide,&amp;nbsp;which is released by the nerves of the gastric mucosa&amp;nbsp;during vagal stimulation.
*The primary actions of&amp;nbsp;gastrin are :
(1) stimulation of gastric acid </atom:summary><link>http://ourphysiology.blogspot.com/2010/02/hormonal-control-of-gastrointestinal.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6587352237198893122</guid><pubDate>Thu, 28 Jan 2010 12:01:00 +0000</pubDate><atom:updated>2010-01-28T04:01:40.408-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">comedk physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">factors affecting les pressure</category><category domain="http://www.blogger.com/atom/ns#">factors affecting lower esophageal sphincter pressure</category><category domain="http://www.blogger.com/atom/ns#">gastrointestinal physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">simethicone</category><title>32 - Factors affecting Lower Esophageal Sphincter (LES) pressure</title><atom:summary type="text">







</atom:summary><link>http://ourphysiology.blogspot.com/2010/01/32-factors-affecting-lower-esophageal.html</link><media:thumbnail xmlns:media="http://search.yahoo.com/mrss/" height="72" url="https://blogger.googleusercontent.com/img/b/R29vZ2xl/AVvXsEjQSWHGqoFXUSepr_JlPqu_SBVC5IiPjlnV6K5CPtUacdObgvZm-IZ0il8Yy0Nj1wZeWp1E8DDPVaxkN5fvXrXE1Ual36oe4m4s8m0HbumdH7XXXXtirntx_fYLpor-jhq2nPOFU4yZn6M/s72-c/factors_affecting_les_pressure11.jpg" width="72"/><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-5955098060161816948</guid><pubDate>Mon, 25 Jan 2010 16:50:00 +0000</pubDate><atom:updated>2010-01-25T09:04:36.015-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">agraphia</category><category domain="http://www.blogger.com/atom/ns#">alexia</category><category domain="http://www.blogger.com/atom/ns#">aphasia</category><category domain="http://www.blogger.com/atom/ns#">aphemia</category><category domain="http://www.blogger.com/atom/ns#">broca's aphasia</category><category domain="http://www.blogger.com/atom/ns#">conductive aphasia</category><category domain="http://www.blogger.com/atom/ns#">global aphasia</category><category domain="http://www.blogger.com/atom/ns#">wernicke's aphasia</category><title>31 - Aphasia</title><atom:summary type="text">























The major language centers of the brain. The motor and sensory areas are presented as landmarks. Interconnecting functional pathways are indicated by letters: A) The connection between Wernicke's and Broca's areas, mediating expression of language utterances in speech; B) The connection between Broca's area and the primary motor area; C) Connection between primary auditory</atom:summary><link>http://ourphysiology.blogspot.com/2010/01/31-aphasia.html</link><media:thumbnail xmlns:media="http://search.yahoo.com/mrss/" height="72" url="https://blogger.googleusercontent.com/img/b/R29vZ2xl/AVvXsEiTV2T_1X9vhDJHaasaoyDUKxADsCTobHSJB-2J4jaPNb5Hre3gkdraiAtUT2SGxco98_GgMhSPkudlylm1OuAEvuaOi6g3xrLWV2Nl2q3fTI4sNU8wi9HPA_ETNRxBGi86v4OulAIR-ug/s72-c/language_centers_of_brain.png" width="72"/><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-1482146818900739969</guid><pubDate>Mon, 25 Jan 2010 11:45:00 +0000</pubDate><atom:updated>2010-01-25T03:45:35.258-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">cerebellum physiology</category><category domain="http://www.blogger.com/atom/ns#">fastigi nucleus</category><category domain="http://www.blogger.com/atom/ns#">globase and emboliform nuclei</category><category domain="http://www.blogger.com/atom/ns#">neocerebellum</category><category domain="http://www.blogger.com/atom/ns#">parts of cerebellum</category><category domain="http://www.blogger.com/atom/ns#">spinocerebellum</category><category domain="http://www.blogger.com/atom/ns#">vestibulo-cerebellum</category><title>30 - Cerebellum</title><atom:summary type="text">*Cerebellum is mainly divided into three parts :
1. Vestibulo cerebellum (Flocculonodular lobe) : Connected to Vestibular nucleus in Brainstem directly .
2. Spinocerebellum : It is divided into 2 parts :
- Medial portion : Fastigi nucleus - Brainstem (so connected indirectly)
- Lateral portion : Globase and Emboliform nuclei - Brainstem (so connected indirectly)
3. Neo-cerebellum :
- Dentate </atom:summary><link>http://ourphysiology.blogspot.com/2010/01/30-cerebellum.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-7002889813077379941</guid><pubDate>Fri, 01 Jan 2010 11:05:00 +0000</pubDate><atom:updated>2010-01-01T03:05:45.526-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">chromosomes mcqs</category><category domain="http://www.blogger.com/atom/ns#">physiology mcqs from past medical pg entrance papers</category><category domain="http://www.blogger.com/atom/ns#">physiology mock test 1</category><title>29 - Physiology mock test 1</title><atom:summary type="text">1)In Wallerian degeneration of a peripheral nerve :

a) The axons regenerate at a rate of 1 cm / day.
b) Occurs in the distal portion of the broken axon leaving an empty tubule 
c) Is a feature of neuropraxia 
d) All the above 

2) Surfactant is a substance that :
a) Is produced in the liver of new borns 
b) Is important  in the new borns but is of little importance in adults
c) Is produced in </atom:summary><link>http://ourphysiology.blogspot.com/2010/01/29-physiology-mock-test-1.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-4543072455206641813</guid><pubDate>Sun, 13 Dec 2009 14:16:00 +0000</pubDate><atom:updated>2009-12-13T06:16:35.740-08:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">normal pressures in the heart and great vessels</category><category domain="http://www.blogger.com/atom/ns#">normal pressures of atria and ventricles</category><category domain="http://www.blogger.com/atom/ns#">normal pressures of pulmonary artery</category><category domain="http://www.blogger.com/atom/ns#">normal pressures of the cardiovascular system</category><title>28 - Normal pressures in the Heart and Great vessels</title><atom:summary type="text">

Normal Pressures in the Heart and Great Vessels
Type of Pressure
Average (mm Hg)
Range (mm Hg)

Right atrium
3
0–8

Right ventricle
&amp;nbsp;&amp;nbsp;
Peak-systolic
25
15–30

End-diastolic
4
0–8

Pulmonary artery
&amp;nbsp;&amp;nbsp;
Mean
15
9–16

Peak-systolic
25
15–30

End-diastolic
9
4–14

Pulmonary artery occlusion
&amp;nbsp;&amp;nbsp;
Mean
9
2–12

Left atrium
&amp;nbsp;&amp;nbsp;
Mean
8
2–12

A wave
10
4–16

V wave
13
</atom:summary><link>http://ourphysiology.blogspot.com/2009/12/28-normal-pressures-in-heart-and-great.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-7316903670809661425</guid><pubDate>Mon, 05 Oct 2009 11:21:00 +0000</pubDate><atom:updated>2009-10-05T04:23:55.683-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">anti diuretic hormone receptors</category><category domain="http://www.blogger.com/atom/ns#">endocrinal physiology</category><category domain="http://www.blogger.com/atom/ns#">v1a receptor</category><category domain="http://www.blogger.com/atom/ns#">v1b receptor</category><category domain="http://www.blogger.com/atom/ns#">v2 receptor</category><category domain="http://www.blogger.com/atom/ns#">vasopressin action on various organs</category><category domain="http://www.blogger.com/atom/ns#">vasopressin receptors</category><title>27 - Vasopressin Receptors</title><atom:summary type="text">*The cellular effects of vasopressin (ADH) are mediated mainly by interactions   of the hormone with the three types of receptors, V1a, V1b,   and V2. 

*The V1a receptor is the most widespread   subtype of Vasopressin receptor; it is found in vascular smooth muscle,   the adrenal gland, myometrium, the bladder, adipocytes, hepatocytes,   platelets, renal medullary interstitial cells, vasa recta </atom:summary><link>http://ourphysiology.blogspot.com/2009/10/27-vasopressin-receptors.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-5302052155949707612</guid><pubDate>Sun, 06 Sep 2009 14:46:00 +0000</pubDate><atom:updated>2009-09-06T07:46:33.001-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">erectile dysfunction physiology</category><category domain="http://www.blogger.com/atom/ns#">nocturnal penile erection</category><category domain="http://www.blogger.com/atom/ns#">nocturnal penile tumescence</category><category domain="http://www.blogger.com/atom/ns#">npt</category><title>26 - Nocturnal Penile Tumescence ( NPT )</title><atom:summary type="text">Nocturnal penile tumescence (NPT) is the spontaneous occurrence of an erection of the penis during sleep. All men without physiological erectile dysfunction experience this phenomenon, usually three to five times during the night. It typically happens during REM sleep. NPT has been given numerous colloquial names which are typically related to the first time the erection is discovered, namely </atom:summary><link>http://ourphysiology.blogspot.com/2009/09/26-nocturnal-penile-tumescence-npt.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6487082107262360904</guid><pubDate>Mon, 08 Jun 2009 06:57:00 +0000</pubDate><atom:updated>2009-06-07T23:57:31.698-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">aiims may 2006 physiology mcqs with answers part 2</category><category domain="http://www.blogger.com/atom/ns#">aiims physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">cushing's triad</category><category domain="http://www.blogger.com/atom/ns#">electromyography mcqs</category><category domain="http://www.blogger.com/atom/ns#">prostate mcqs</category><category domain="http://www.blogger.com/atom/ns#">rem sleep and wakefulness differences</category><title>25 - AIIMS MAY 2006 mcqs with answers part 2</title><atom:summary type="text">6q: which of the following organs secrete zinc in large amounts in man ?


a. seminal vesicle
b. prostate
c. epididymis
d. vas


 answer c. prostate  
 
 7q: follicular stimulating hormone receptors are present on ?
 
 a. theca cells
 b. granulosa cells
 c. leydig cells
 d. basement membrane of ovarian follicle
 
 
 answer b. granulosa cells 
 
 8q: the main difference between REM sleep and </atom:summary><link>http://ourphysiology.blogspot.com/2009/06/25-aiims-may-2006-mcqs-with-answers.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-7631446196959420839</guid><pubDate>Mon, 08 Jun 2009 06:53:00 +0000</pubDate><atom:updated>2009-06-07T23:53:22.153-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">aiims may 2006 physiology mcqs with answers part 1</category><category domain="http://www.blogger.com/atom/ns#">aiims physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">oxygen hemoglobin dissociation curve mcqs</category><category domain="http://www.blogger.com/atom/ns#">second messengers mcqs</category><category domain="http://www.blogger.com/atom/ns#">vanilloid receptors</category><title>24 - AIIMS MAY 2006 mcqs with answers part 1</title><atom:summary type="text">1q: vanilloid receptors are activated by ?


a. pain
b. vibration
c. touch
d. pressure




 answer a. pain   
 
 2q: the sodium-potassium pump is an example of ?
 
 a. active transport
 b. passive transport
 c. facilitated diffusion
 d. osmosis
 
 
 answer a. active transport  
 
 3q: which one of the following acts as a second messenger ?
 
 a. Mg++
 b. Cl-
 c. Ca++
 d. PO4+3
 
 
 answer c . Ca </atom:summary><link>http://ourphysiology.blogspot.com/2009/06/24-aiims-may-2006-mcqs-with-answers.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6797412231935756856</guid><pubDate>Thu, 30 Apr 2009 05:33:00 +0000</pubDate><atom:updated>2009-04-29T22:33:44.285-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">calculation of faecal osmolality</category><category domain="http://www.blogger.com/atom/ns#">faecal osmolality</category><category domain="http://www.blogger.com/atom/ns#">faecal osmolar gap</category><category domain="http://www.blogger.com/atom/ns#">osmolality article</category><category domain="http://www.blogger.com/atom/ns#">osmolality mcqs</category><category domain="http://www.blogger.com/atom/ns#">osmotic diarrhea</category><title>23 - Faecal osmolar gap</title><atom:summary type="text">

Faecal osmolality is used to determine the faecal osmolar gap. This is defined as the difference between the measured osmolality and an osmolality calculated from 2 x (Na+K).


&amp;nbsp;If the gap is greater than 100 mmol/L this is consistent with an osmotic diarrhoea (eg carbohydrate (poor absorption, eg mannitol, sorbitol, lactulose); monosaccharides; short chain fatty acids; magnesium as used </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/23-faecal-osmolar-gap.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-4969701859272871037</guid><pubDate>Thu, 30 Apr 2009 05:32:00 +0000</pubDate><atom:updated>2009-04-29T22:32:20.500-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">differential diagnosis of hypernatremia and hyponatremia</category><category domain="http://www.blogger.com/atom/ns#">osmolality article</category><category domain="http://www.blogger.com/atom/ns#">renal failure mcqs</category><category domain="http://www.blogger.com/atom/ns#">siadh identification</category><category domain="http://www.blogger.com/atom/ns#">urine osmolality</category><title>22 - Urine Osmolality</title><atom:summary type="text">Urine osmolality is an important test for the concentrating ability of the kidney. Interpretation of urine osmolality must always be made in the light of the appropriate physiological response to the state of hydration of the patient. The test is useful in the following areas:
For determining the      differential diagnosis of hyper- or hyponatraemia.


For identifying SIADH      (urine </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/22-urine-osmolality.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-3447679172687261167</guid><pubDate>Thu, 30 Apr 2009 05:30:00 +0000</pubDate><atom:updated>2009-04-29T22:30:16.830-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">causes of increased osmolar gap</category><category domain="http://www.blogger.com/atom/ns#">osmolality article</category><category domain="http://www.blogger.com/atom/ns#">osmolality mcqs</category><category domain="http://www.blogger.com/atom/ns#">serum osmolality calculation formula</category><category domain="http://www.blogger.com/atom/ns#">serum osmolar gap</category><title>21 - Serum Osmolar gap</title><atom:summary type="text">The osmolar gap is determined by subtracting the calculated osmolality from the measured osmolality. While there are many formulae for the calculated osmolality, the most commonly used is:


Calculated osmolality = 2 x serum sodium + serum glucose + serum urea (all in mmol/L).


The normal osmolar gap is up to 10 mmol/L and values in excess of this usually indicate the presence of an exogenous </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/21-serum-osmolar-gap.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-2500615421758180387</guid><pubDate>Thu, 30 Apr 2009 05:28:00 +0000</pubDate><atom:updated>2009-04-29T22:28:13.008-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">causes of increased osmolality</category><category domain="http://www.blogger.com/atom/ns#">hyponatremia investigation</category><category domain="http://www.blogger.com/atom/ns#">osmolality article</category><category domain="http://www.blogger.com/atom/ns#">osmolality mcqs</category><category domain="http://www.blogger.com/atom/ns#">serum osmolality</category><category domain="http://www.blogger.com/atom/ns#">true hyponatremia</category><title>20 - Serum Osmolality</title><atom:summary type="text">

Serum osmolality is a useful preliminary investigation for identifying the cause of hyponatraemia. If a patient with significant hyponatraemia (serum sodium &amp;lt; 130 mmol/L) has a normal plasma osmolality, the patient may have pseudohyponatraemia due to excess lipids or proteins, or the sample may have been collected from a drip arm containing dextrose.&amp;nbsp;


If the patient has an increased </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/20-serum-osmolality.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-3747611424978237670</guid><pubDate>Thu, 30 Apr 2009 05:26:00 +0000</pubDate><atom:updated>2009-04-29T22:26:26.045-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">faecal osmolality</category><category domain="http://www.blogger.com/atom/ns#">hyponatremia investigation</category><category domain="http://www.blogger.com/atom/ns#">osmolality article</category><category domain="http://www.blogger.com/atom/ns#">osmolality clinical uses</category><category domain="http://www.blogger.com/atom/ns#">osmolality mcqs</category><category domain="http://www.blogger.com/atom/ns#">osmolality uses</category><category domain="http://www.blogger.com/atom/ns#">osmolar gap identification</category><title>19 - Osmolality clinical uses</title><atom:summary type="text">

Serum osmolality is used in two main circumstances: investigation of hyponatraemia and identification of an osmolar gap. Urine osmolality is an important test of renal concentrating ability, for identifying disorders of the ADH mechanism, and identifying causes of hyper-or hyponatraemia. Faecal osmolality can be used to assist with diagnosis of the cause of diarrhoea.



</atom:summary><link>http://ourphysiology.blogspot.com/2009/04/19-osmolality-clinical-uses.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6443243118355069674</guid><pubDate>Thu, 30 Apr 2009 05:24:00 +0000</pubDate><atom:updated>2009-04-29T22:24:20.077-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">osmolality and ADH physiology</category><category domain="http://www.blogger.com/atom/ns#">osmolality and kidney physiology</category><category domain="http://www.blogger.com/atom/ns#">osmolality calculating formula</category><category domain="http://www.blogger.com/atom/ns#">osmolality measurement</category><category domain="http://www.blogger.com/atom/ns#">osmometer</category><title>18 - Osmolality measurement</title><atom:summary type="text">The osmolality of a solution can be measured using an osmometer. The most commonly used instrument in modern laboratories is a freezing point depression osmometer. This instrument measures the change in freezing point that occurs in a solution with increasing osmolality. Osmolality can be measured in samples of serum (gold top tube) or heparin plasma (lime top tube).


Plasma osmolality can also </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/18-osmolality-measurement.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6926871565009989634</guid><pubDate>Thu, 30 Apr 2009 05:22:00 +0000</pubDate><atom:updated>2009-04-29T22:22:56.454-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">osmolality and ADH relation</category><category domain="http://www.blogger.com/atom/ns#">osmolality and kidney physiology</category><category domain="http://www.blogger.com/atom/ns#">osmolality article</category><category domain="http://www.blogger.com/atom/ns#">osmolality physiology</category><category domain="http://www.blogger.com/atom/ns#">urine osmolality</category><title>17 - Osmolality physiology</title><atom:summary type="text">The osmolality of plasma is closely regulated by anti-diuretic hormone (ADH). In response to even small increases in plasma osmolality (usually rises in plasma sodium), ADH release from the pituitary is increased causing water resorption in the distal tubules and collecting ducts of the kidney and correction of the increased osmolality. The opposite happens in response to a low plasma osmolality </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/17-osmolality-physiology.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-5513432786074419034</guid><pubDate>Thu, 30 Apr 2009 05:20:00 +0000</pubDate><atom:updated>2009-04-29T22:20:48.415-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">avogadro's number</category><category domain="http://www.blogger.com/atom/ns#">osmolality and ADH</category><category domain="http://www.blogger.com/atom/ns#">Osmolality introduction</category><category domain="http://www.blogger.com/atom/ns#">osmolality mcqs</category><category domain="http://www.blogger.com/atom/ns#">osmolality physiology</category><category domain="http://www.blogger.com/atom/ns#">units of osmolality</category><title>16 - Osmolality introduction</title><atom:summary type="text">Osmolality is a count of the number of particles in a fluid sample. The unit for counting is the mole which is equal to 6.02 x 1023&amp;nbsp;particles (Avogadro's Number). Molarity is the number of particles of a particular substance in a volume of fluid (eg mmol/L) and molality is the number of particles disolved in a mass weight of fluid (mmol/kg). Osmolality is a count of the total number of </atom:summary><link>http://ourphysiology.blogspot.com/2009/04/16-osmolality-introduction.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-581892609686015760</guid><pubDate>Mon, 20 Apr 2009 07:17:00 +0000</pubDate><atom:updated>2009-04-20T03:28:15.068-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">basal metabolic rate mcqs</category><category domain="http://www.blogger.com/atom/ns#">factors affecting basal metabolic rate</category><category domain="http://www.blogger.com/atom/ns#">factors influencing bmr</category><category domain="http://www.blogger.com/atom/ns#">pgi chandigarh 2001 physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">pgimer chandigarh 2001 december paper mcqs with answers</category><title>15 - Basal Metabolic Rate Mcqs with notes</title><atom:summary type="text">1q: which of the following statements is true regarding basal metabolic rate ?


a. increased in starvation
b. it is not influenced by hormonal changes
c. it is not affected by dietary changes
d. decreased by 40 % in starvation
e. it is not affected by energy expenditure 


  answer  only  ‘d’ is the correct statement   

Some important points about basal metabolic rate :


1. the energy expended</atom:summary><link>http://ourphysiology.blogspot.com/2009/04/15-basal-metabolic-rate-mcqs-with-notes.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-6516104997498389440</guid><pubDate>Fri, 27 Mar 2009 10:29:00 +0000</pubDate><atom:updated>2009-03-27T03:30:47.507-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">aiims may 2005 physiology mcqs with answers</category><category domain="http://www.blogger.com/atom/ns#">endocrinology mcqs</category><category domain="http://www.blogger.com/atom/ns#">measurement of body fluid volumes</category><category domain="http://www.blogger.com/atom/ns#">peptide hormones mcqs</category><category domain="http://www.blogger.com/atom/ns#">transportor binding proteins</category><title>14 - AIIMS may 2005 physiology mcqs with answers part 3</title><atom:summary type="text">10q: there is a mutation of gene coding for the ryanodine receptors in malignant hyperthermia . which of the following statements best explains the increased heat production in malignant hyperthermia ?a. increased muscle metabolism by excess of calcium ionsb. thermic effect of foodc. increased sympathetic discharged. mitochondrial thermogenesis  answer  a. increased muscle metabolism by excess of</atom:summary><link>http://ourphysiology.blogspot.com/2009/03/14-aiims-may-2005-physiology-mcqs-with.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-7628884103500418845</guid><pubDate>Fri, 27 Mar 2009 10:27:00 +0000</pubDate><atom:updated>2009-03-27T03:29:01.608-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">aiims may 2005 physiology mcqs with answers</category><category domain="http://www.blogger.com/atom/ns#">digestive enzymes physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">lung physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">respiratory system physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">veins mcqs</category><title>13 - AIIMS may 2005 physiology mcqs with answers part 2</title><atom:summary type="text">6q: all of the following enzymes are active with in a cell except?a. trypsinb. fumarasec. exokinased. alcohol dehydrogenase  answer  a. trypsin   7q: exercise is also prescribed as an adjuvant treatment for depression . most probably this acts by ?a. increasing pulse pressureb. improving hemodynamicsc. raising endorphin levelsd. inducing good sleep  answer  c. raising endorphin levels   8q: the </atom:summary><link>http://ourphysiology.blogspot.com/2009/03/13-aiims-may-2005-physiology-mcqs-with.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item><item><guid isPermaLink="false">tag:blogger.com,1999:blog-2649822511498143527.post-625243194334922900</guid><pubDate>Fri, 27 Mar 2009 10:18:00 +0000</pubDate><atom:updated>2009-03-27T03:31:16.970-07:00</atom:updated><category domain="http://www.blogger.com/atom/ns#">aiims may 2005 physiology mcqs with answers</category><category domain="http://www.blogger.com/atom/ns#">ecg physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">insulin physiology mcqs</category><category domain="http://www.blogger.com/atom/ns#">law of projection</category><category domain="http://www.blogger.com/atom/ns#">phantom limb</category><title>12 - AIIMS may 2005 physiology mcqs with answers part 1</title><atom:summary type="text">1q: group A nerve fibers are more susceptible to ?a. pressureb. hypoxiac. local anaestheticsd. temperature  answer  a. pressure   2q: in a fetus the insulin secretion begins by ?a. 3 monthsb. 5 monthsc. 7 monthsd. 9 months  answer  a . 3 months   3q: phantom limb sensations are best described by ?a. weber Fechner lawb. power lawc. bell magendie lawd. law of projection  answer  d. law of </atom:summary><link>http://ourphysiology.blogspot.com/2009/03/12-aiims-may-2005-physiology-mcqs-with.html</link><thr:total>0</thr:total><author>prashanthparigela@gmail.com (doctor)</author></item></channel></rss>